Metrnl cDNA ORF clone, Mus musculus(house mouse)

Adipocyte Metrnl Antagonizes Insulin Resistance Through PPAR? Signaling.
Li ZY, Song J, Zheng SL, Fan MB, Guan YF, Qu Y, Xu J, Wang P, Miao CY
Diabetes64(12)4011-22(2015 Dec)

Data (including data from studies in transgenic/knockout mice and 3T3-L1 cells) suggest expression of Metrnl in white adipocytes controls insulin sensitivity via activation of PPARg (peroxisome proliferator activated receptor gamma) signaling pathway.
Title: Adipocyte Metrnl Antagonizes Insulin Resistance Through PPAR? Signaling.

Metrnl represents a novel cytokine, which is likely involved in both innate and acquired immune responses.
Title: METEORIN-LIKE is a cytokine associated with barrier tissues and alternatively activated macrophages.

Study reports the identification of meteorin-like (Metrnl), a circulating factor that is induced in muscle after exercise and in adipose tissue upon cold exposure; increasing circulating levels of Metrnl stimulates energy expenditure and improves glucose tolerance and the expression of genes associated with beige fat thermogenesis and anti-inflammatory cytokines.
Title: Meteorin-like is a hormone that regulates immune-adipose interactions to increase beige fat thermogenesis.

Results show that Subfatin is a novel adipokine regulated by adipogenesis and obesity, with tissue distribution different from its homologue Meteorin
Title: Subfatin is a novel adipokine and unlike Meteorin in adipose and brain expression.

Cometin is a novel neurotrophic factor(meteorin protein family) that promotes neurite outgrowth and neuroblast migration in vitro and supports survival of spiral ganglion neurons in vivo.
Title: Cometin is a novel neurotrophic factor that promotes neurite outgrowth and neuroblast migration in vitro and supports survival of spiral ganglion neurons in vivo.