EAAT3 Antibody, mAb, Rabbit
EAAT3 Antibody, MAb, Rabbit

Western blot analysis of EAAT3 expression in Human fetal brain lysate.

EAAT3 Antibody, mAb, Rabbit

Transports L-glutamate and also L- and D-aspartate. Essential for terminating the postsynaptic action of glutamate by rapidly removing released glutamate from the synaptic cleft. Acts as a symport by cotransporting sodium. Negatively regulated by ARL6IP5.
A04791
¥37,309.00

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Overview
Specificity This product is specific to EAAT3
Host Species Rabbit
Immunogen A synthesized peptide derived from human EAAT3
Species Reactivity Human; Mouse; Rat
Conjugate Unconjugated

Applications
Working concentrations for specific applications should be determined by the investigators. The appropriate concentrations may be affected by secondary antibody affinity, antigen concentration, the sensitivity of the method of detection, temperature, the length of the incubations, and other factors. The suitability of this antibody for applications other than those listed below has not been determined. The following concentration ranges are recommended starting points for this product.
Application Recommended Usage
Western Blot 1:1000-1:2000
Immunohistochemistry (IHC) 1:100-1:200
Immunocytochemistry/Immunofluorescence (ICC/IF) 1:50-1:200
Immunohistochemistry Frozen (IHF) 1:50-1:200

Properties
Form Liquid
Storage Buffer Supplied in 10mM phosphate buffered saline , pH 7.4, 150mM sodium chloride, 0.05% BSA, 0.02% sodium azide and 50% glycerol.
Storage Instructions Store at -20°C. This product is stable for 1 year upon receipt, when handled and stored as instructed.
Purification Affinity-chromatography
Isotype Rabbit IgG
Clonality Monoclonal

Examples
  • EAAT3 Antibody, MAb, Rabbit
    • EAAT3 Antibody, MAb, Rabbit

    Western blot analysis of EAAT3 expression in Human fetal brain lysate.


Background
Target Background Transports L-glutamate and also L- and D-aspartate. Essential for terminating the postsynaptic action of glutamate by rapidly removing released glutamate from the synaptic cleft. Acts as a symport by cotransporting sodium. Negatively regulated by ARL6IP5.
Synonyms SLC1A1, EAAC1, EAAT3, Eaac-1;

For research use only. Not intended for human or animal clinical trials, therapeutic or diagnostic use.